Showing posts with label research. Show all posts
Showing posts with label research. Show all posts

Thursday, November 10

This is some more info about nitrit, a E 250 substance found in processed meat (and smoked meat/fish)
Its a preservative, color enhancer and keep botulism (a disease) at bay. For a Swedish Article about this

Background: Humans are exposed to preformed N-nitroso compounds (NOCs) and endogenous NOCs. Several NOCs are potential human carcinogens, including N-nitrosodimethylamine (NDMA), but evidence from population studies is inconsistent.
Objective: Weexamined therelation between dietaryNOCs(NDMA), the endogenous NOC index, and dietary nitrite and cancer incidence in the European Prospective Investigation into Cancer and Nutrition (EPIC)–Norfolk, United Kingdom, study.
Design: This was a prospective study of 23,363 men and women, aged 40–79 y, who were recruited in 1993–1997 and followed up to 2008. The baseline diet was assessed with food-frequency questionnaires.
Results: There were 3268 incident cancers after a mean follow-up of 11.4 y. Dietary NDMA intake was significantly associated with increased cancer risk in men and women [hazard ratio (HR): 1.14; 95% CI: 1.03, 1.27; P for trend = 0.03) and in men (HR: 1.24; 95% CI: 1.07, 1.44; P for trend = 0.005) when the highest quartile was comparedwith the lowest quartile in age- and sex-adjusted analyses but not in multivariate analyses (HR: 1.10; 95% CI: 0.97, 1.24; HR for men: 1.18; 95% CI: 1.00, 1.40; P for trend ≥ 0.05). When continuously analyzed, NDMA was associated with increased risk of gastrointestinal cancers (HR: 1.13; 95% CI: 1.00, 1.28), specifically of rectal cancer (HR: 1.46; 95% CI: 1.16, 1.84) per 1-SD increase after adjustment for age, sex, body mass index, cigarette smoking status, alcohol intake, energy intake, physical activity, education, and menopausal status (in women). The endogenous NOC index and dietary nitrite were not significantly associated with cancer risk. There was a significant interaction between plasma vitamin C concentrations and dietary NDMA intake on cancer incidence (P for interaction < 0.00001).
Conclusions: Dietary NOC (NDMA) was associated with a higher gastrointestinal cancer incidence, specifically of rectal cancer. Plasma vitamin C may modify the relation between NDMA exposure and cancer risk.
N-nitroso compounds and cancer incidence: the European Prospective Investigation into Cancerand Nutrition (EPIC)–Norfolk study Am J Clin Nutr March 23, 2011.

Friday, April 8

Found this extremely interesting study at kollo! Sleep deprivation and calorie usage.



Background: Epidemiologic evidence indicates that chronic sleep curtailment increases risk of developing obesity, but the mechanisms behind this relation are largely unknown.

Objective: We examined the influence of a single night of total sleep deprivation on morning energy expenditures and food intakes in healthy humans.

Design: According to a balanced crossover design, we examined 14 normal-weight male subjects on 2 occasions during a regular 24-h sleep-wake cycle (including 8 h of nocturnal sleep) and a 24-h period of continuous wakefulness. On the morning after regular sleep and total sleep deprivation, resting and postprandial energy expenditures were assessed by indirect calorimetry, and the free-choice food intake from an opulent buffet was tested in the late afternoon at the end of the experiment. Circulating concentrations of ghrelin, leptin, norepinephrine, cortisol, thyreotropin, glucose, and insulinwere repeatedly measured over the entire 24-h session.

Results: In comparison with normal sleep, resting and postprandial energy expenditures assessed on the subsequent morning were significantly reduced after sleep deprivation by ≈5% and 20%, respectively (P < 0.05 and P < 0.0001). Nocturnal wakefulness increased morning plasma ghrelin concentrations (P < 0.02) and nocturnal and daytime circulating concentrations of thyreotropin, cortisol, and norepinephrine (P < 0.05) as well as morning postprandial plasma glucose concentrations (P < 0.05). Changes in food intakes were variable, and no differences between wake and sleep conditions were detected. Conclusion: Our findings show that one night of sleep deprivation acutely reduces energy expenditure in healthy men, which suggests that sleep contributes to the acute regulation of daytime energy expenditure in humans

Full text here

Tuesday, March 8

Interesting read

Athletes and exercisers have utilised high-protein diets for centuries. The objective of this review is to examine the evidence for the efficacy and potential dangers of high-protein diets. One important factor to consider is the definition of a 'high-protein diet'. There are several ways to consider protein content of a diet. The composition of the diet can be determined as the absolute amount of the protein (or other nutrient of interest), the % of total energy (calories) as protein and the amount of protein ingested per kg of body weight. Many athletes consume very high amounts of protein. High-protein diets most often are associated with muscle hypertrophy and strength, but now also are advocated for weight loss and recovery from intense exercise or injuries. Prolonged intake of a large amount of protein has been associated with potential dangers, such as bone mineral loss and kidney damage. In otherwise healthy individuals, there is little evidence that high protein intake is dangerous. However, kidney damage may be an issue for individuals with already existing kidney dysfunction. Increased protein intake necessarily means that overall energy intake must increase or consumption of either carbohydrate or fat must decrease. In conclusion, high protein intake may be appropriate for some athletes, but there are potential negative consequences that must be carefully considered before adopting such a diet. In particular, care must be taken to ensure that there is sufficient intake of other nutrients to support the training load. 

Proc Nutr Soc. 2011 Mar 7:1-10. Tipton KD. Efficacy and consequences of very-high-protein diets for athletes and exercisers.  (found it at kollo)

Sunday, November 7

The competition diet.
I feel I want to share the diet and some thought and the reasoning why I have done what I have done with it.

I started dieting in July. the first 2 months I did 1800 kcal a day with an eating day. I did however do morning walks (was told not to at the start of the diet).
I did lose some bodyfat but not nearly enough in my opinion so I figured that I needed to step up the current diet (but no dramas, I knew what I had to do).
After I got sick, in the end of August, I started doing cyclic dieting. I lowered my calories to 1700 on training days and 16-1400 on resting days. I kept a eating day, but limited to 2500 kcal. I increased the morning walk length a bit (75min), I started taking extra walks on resting days with my friends, in the forest and on tracks. I also increased the weekends walks to 90 min. the ex coach also increased the cardio to 30 min spin/bike riding at training days. And I did so.

I increased my protein to 4g per kg body weight, kept the fat to 45-60g per kg bw a days and lowered the carbs to fill out the rest. Over these 2 months I got down to where I am now.
The diet has been based around fibrous vegetables, lean chicken, egg, 1 or 2 (17 g each) flat thin bread, low fat cheese, low fat cottage cheese, low fat quark, milk, protein powder, amino acids, rice cakes (after gym) and loads of vitamins and 6-8 liters of water a day.

On eating day, that I prefer to call carb up day or re-feed. I have lowered my protein to about 2 g /per kg body weight, kept fats low (30g kg bw /day) and increased carbs to around 300 g /kg bw. I have had dinners out with friends and family and sometimes been eating a little more than 2500 and sometimes less. I havent always done a proper re-feed but for the most part this is it.
I have kept the main part of the re-feeds to natural food sources.
I have had oats, potato, wholemeal bread and berries as a main source of carbs.

The reasoning behind the re-feeds, to increase the hormone leptin that drops when you diet. this hormone is boosted by an temporary increase in carbs.
http://www.leangains.com/2010/03/intermittent-fasting-set-point-and.html
http://www.ncbi.nlm.nih.gov/pubmed/11126336?ordinalpos=47&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsP anel.Pubmed_RVDocSum

CONCLUSIONS: CHO OF (carbohydrate overfeeding), but not FAT OF, increases energy expenditure and leptin concentration.

The berries are good for replenishing your liver glycogen and helps with hunger, this is especially noticeable in women. Thats why Ive eaten the berries.

Oats have great health benefits, on the mood and on the body
http://www.elements4health.com/oats.html

I have had milk produces because they increase the fat oxidation in the body
Zemel M B, Shi H. Greer B, DiRienzo D & Zemel PC. Regulation of adiposity by dietary calcium FASEB J. 14:1132-1138, 2000.

Zemel M B, Thompson W, Zemel P, Nocton A M, et al. Dietary calcium and dairy products accelerate weight and fat loss during energy restriction in obese adults Am J Clin Nutr 75(suppl. 2):a342S, 2000.

Zemel MB. Mechanisms of dairy modulation of adiposity J Nutr 133:252S-256S, 2003.

Teegarden D. Calcium intake and reduction in weight or fat mass. J Nutr 133:249S-251S, 2003.

E L Melanson et al., Relation between calcium intake and fat oxidation in adult humans. Int Journal of Obesity 27:196-203, 2003.

A diet high in protein increases the chance of maintaining as much muscle as possible when dieting. It also helps feeling fuller (and is tasty) - there are loads of research showing this so I wont put them in here.

Fibrous greens fills you up and keeps the body working. The fibers/carbs in the vegetables is hard for the body to take up and they are boosted with great vitamin's.
They also do other great things for your body
http://newsroom.ucla.edu/portal/ucla/ucla-study-reports-broccoli-may-46578.aspx

Eggs are a great source of healthy fats and protein and helps you feel fuller for longer. (swedish) http://traningslara.se/agg-ett-fantastiskt-livsmedel/

Ill probably write some more about this as Ive probably forgotten things but this is the main part of the diet. Now Im only focusing to stay where I am, keep the same foods and train hard!

Friday, October 29

Interesting research!
BACKGROUND: Fatty acids (FAs) may be important dietary components that modulate osteoporotic fracture risk.

OBJECTIVE: The objective was to examine FA intake in relation to osteoporotic fractures.


DESIGN: The participants were postmenopausal women enrolled in the Women's Health Initiative (n = 137,486). Total fractures were identified by self-report; hip fractures were confirmed by medical record review. FA intake was estimated from baseline food-frequency questionnaires and standardized to total caloric intake. No data on omega-3 (n-3) FA supplements were available. Cox proportional hazard models were constructed to estimate risk of fracture.


RESULTS: Higher saturated FA consumption was associated with higher hip fracture risk [quartile 4 multivariate-adjusted hazard ratio (HR): 1.31; 95% CI: 1.11, 1.55; P for trend = 0.001]. Lower total fracture risk was associated with a higher monounsaturated FA intake (quartile 3 HR: 0.94; 95% CI: 0.89, 0.98; P for trend = 0.050) and polyunsaturated FA intake (quartile 4 HR: 0.95; 95% CI: 0.90, 0.99; P for trend = 0.019). Unexpectedly, higher consumption of marine n-3 FAs was associated with greater total fracture risk (quartile 4 HR: 1.07; 95% CI: 1.02, 1.12; P for trend = 0.010), whereas a higher n-6 FA intake was associated with a lower total fracture risk (quartile 4 HR: 0.94; 95% CI: 0.89, 0.98; P for trend 0.009).


CONCLUSIONS: These results suggest that saturated FA intake may significantly increase hip fracture risk, whereas monounsaturated and polyunsaturated FA intakes may decrease total fracture risk. In postmenopausal women with a low intake of marine n-3 FAs, a higher intake of n-6 FAs may modestly decrease total fracture risk.
Am J Clin Nutr. 2010 Oct 27. Fatty acid consumption and risk of fracture in the Women's Health Initiative.

http://www.ajcn.org/cgi/rapidpdf/ajcn.2010.29955v1

Wednesday, October 13

On the way to my seamstress! Massive soreness in the entire back section of my body lol love it! On Fridays we have test coffee and you get a week and you bring cookies! My week is this week (I've never had anything and never will) very tempted to bring carrots or something i can or would eat... But they made me promise I would bring something unhealthy... I guess I have to go to the shops then lol

Very interesting study! Indicates howimportant sleep is!

Ann Intern Med. 2010 Oct 5;153(7):435-41.
Insufficient sleep undermines dietary efforts to reduce adiposity.
http://www.annals.org/content/153/7/435.long
BACKGROUND: Sleep loss can modify energy intake and expenditure.

OBJECTIVE: To determine whether sleep restriction attenuates the effect of a reduced-calorie diet on excess adiposity.

DESIGN: Randomized, 2-period, 2-condition crossover study.

SETTING: University clinical research center and sleep laboratory.

PATIENTS: 10 overweight nonsmoking adults (3 women and 7 men) with a mean age of 41 years (SD, 5) and a mean body mass index of 27.4 kg/m² (SD, 2.0).

INTERVENTION: 14 days of moderate caloric restriction with 8.5 or 5.5 hours of nighttime sleep opportunity.

MEASUREMENTS: The primary measure was loss of fat and fat-free body mass. Secondary measures were changes in substrate utilization, energy expenditure, hunger, and 24-hour metabolic hormone concentrations.

RESULTS: Sleep curtailment decreased the proportion of weight lost as fat by 55% (1.4 vs. 0.6 kg with 8.5 vs. 5.5 hours of sleep opportunity, respectively; P = 0.043) and increased the loss of fat-free body mass by 60% (1.5 vs. 2.4 kg; P = 0.002). This was accompanied by markers of enhanced neuroendocrine adaptation to caloric restriction, increased hunger, and a shift in relative substrate utilization toward oxidation of less fat.

LIMITATION: The nature of the study limited its duration and sample size.

CONCLUSION: The amount of human sleep contributes to the maintenance of fat-free body mass at times of decreased energy intake. Lack of sufficient sleep may compromise the efficacy of typical dietary interventions for weight loss and related metabolic risk reduction.

PRIMARY FUNDING SOURCE: National Institutes of Health.

Tuesday, October 5

Aspartame and cancer, some more research indicating that aspartame might cause cancer :(

BACKGROUND: Aspartame (APM) is a well-known intense artificial sweetener used in more than 6,000 products. Among the major users of aspartame are children and women of childbearing age. In previous lifespan experiments conducted on Sprague-Dawley rats we have shown that APM is a carcinogenic agent in multiple sites and that its effects are increased when exposure starts from prenatal life.

OBJECTIVE: The aim of this study is to evaluate the potential of APM to induce carcinogenic effects in mice.


METHODS: Six groups of 62-122 male and female Swiss mice were treated with APM in feed at doses of 32,000, 16,000, 8,000, 2,000, or 0 ppm from prenatal life (12 days of gestation) until death. At death each animal underwent complete necropsy and all tissues and organs of all animals in the experiment were microscopically examined.


RESULTS: APM in our experimental conditions induces in males a significant dose-related increased incidence of hepatocellular carcinomas (P < 0.01), and a significant increase at the dose levels of 32,000 ppm (P < 0.01) and 16,000 ppm (P < 0.05). Moreover, the results show a significant dose-related increased incidence of alveolar/bronchiolar carcinomas in males (P < 0.05), and a significant increase at 32,000 ppm (P < 0.05).


CONCLUSIONS: The results of the present study confirm that APM is a carcinogenic agent in multiple sites in rodents, and that this effect is induced in two species, rats (males and females) and mice (males). No carcinogenic effects were observed in female mice. 

Am J Ind Med. 2010 Sep 30. Aspartame administered in feed, beginning prenatally through life span, induces cancers of the liver and lung in male Swiss mice  

Thursday, July 1

AIM: Post-menopause is linked to an increased risk of death from cardiovascular disease. Physical exercise and healthy dietary habits are normally suggested to enhance health. The aim of this study was to verify whether the time of day of walking had different effects on both spontaneous dietary intake and body composition modification in overweight and sedentary post-menopausal women.

METHODS: Forty-two sedentary post-menopausal women (53.46+/-3.32 yrs) were recruited. Thirty-three completed the study: 29 were suitable for statistical analysis. Of those, 14 walked in the morning (MG) and 15 in the early evening (EG). Body composition, dietary habits and predicted VO2max were investigated. Food intake was analysed for energy, macronutrients and daily distribution.


RESULTS:
The Mann-Whitney test showed that according to the time of day of walking there were different fat mass (FM) reductions and dietary behaviour responses. EG reduced FM greater than MG and showed a major increase in morning energy intake (EI). Sub-samples did not differ in total EI, daily macronutrient portioning and daily meals variations. The variation of FM was correlated with that of proteins (r=-0.352), morning EI (r=-0.367) and aerobic performance (r=0.369). Both MG and EG improved their aerobic performance.

CONCLUSION: The positive effects of walking on health could be optimised by its evening execution because it could also be linked to spontaneous dietary habit modification. J Sports Med Phys Fitness. 2010 Jun;50(2):196-201. Effects of the time of day of walking on dietary behaviour, body composition and aerobic fitness in post-menopausal women. 


J Sports Med Phys Fitness. 2010 Jun;50(2):196-201. Effects of the time of day of walking on dietary behaviour, body composition and aerobic fitness in post-menopausal women. 

Of course you eat less at night if your out walking but the interesting part is the bold one, suggest to me that you have a greater fat metabolism walking in the morning rather than at night. There are no numbers so there is no suggestion as to how much it would differ. But interesting nevertheless!
(from kolozzeum)

Monday, March 15

good morning

more interesting studies
BACKGROUND: Obesity has reached epidemic proportions in the United States. It is implicated in the development of a variety of chronic disease states and is associated with increased levels of inflammation and oxidative stress. The objective of this study is to examine the effect of Medifast's meal replacement program (MD) on body weight, body composition, and biomarkers of inflammation and oxidative stress among obese individuals following a period of weight loss and weight maintenance compared to a an isocaloric, food-based diet (FB).

METHODS: This 40-week randomized, controlled clinical trial included 90 obese adults with a body mass index (BMI) between 30 and 50 kg/m2, randomly assigned to one of two weight loss programs for 16 weeks and then followed for a 24-week period of weight maintenance. The dietary interventions consisted of Medifast's meal replacement program for weight loss and weight maintenance, or a self-selected, isocaloric, food-based meal plan.

RESULTS: Weight loss at 16 weeks was significantly better in the Medifast group (MD) versus the food-based group (FB) (12.3% vs. 6.9%), and while significantly more weight was regained during weight maintenance on MD versus FB, overall greater weight loss was achieved on MD versus FB. Significantly more of the MD participants lost [greater than or equal to]5% of their initial weight at week 16 (93% vs. 55%) and week 40 (62% vs. 30%). There was no difference in satiety observed between the two groups during the weight loss phase. Significant improvements in body composition were also observed in MD participants compared to FB at week 16 and week 40. At week 40, both groups experienced improvements in biochemical outcomes and other clinical indicators.

CONCLUSIONS: Our data suggest that the meal replacement diet plan evaluated was an effective strategy for producing robust initial weight loss and for achieving improvements in a number of health-related parameters during weight maintenance, including inflammation and oxidative stress, two key factors more recently shown to underlie our most common chronic diseases.
Nutr J. 2010 Mar 11;9(1):11. Efficacy of a meal replacement diet plan compared to a food-based diet plan after a period of weight loss and weight maintenance: a randomized controlled trial. 

Saturday, March 13

This is a very good read.

Protein metabolism adapts during caloric restriction (CR) to minimize protein loss, and it is unclear whether greater fat stores favorably affect this response. We sought to determine whether protein metabolism is related to degree of obesity and whether the response to CR is impacted by pre-CR adiposity level. Whole body protein metabolism was studied in 12 obese women over a wide range of BMI (30-53 kg/m(2)) as inpatients using [1-(13)C]leucine as a tracer following 5 days of a weight-maintaining diet and then after 30 days of CR (1,400 kcal deficit with maintained protein intake). When expressed as total rates, per body weight (BW) or per fat-free mass (FFM), leucine rate of appearance (Ra), and nonoxidative leucine disposal (NOLD) were significantly higher in the individuals with a greater degree of obesity (P < 0.05). Leucine oxidation (Rox) was also higher in more highly obese women when expressed as a total rate (P < 0.05) but not if expressed per BW or FFM. CR reduced BW, FFM, and fat mass (P < 0.001), and declines were relatively similar between individuals. CR reduced Ra (P < 0.001), NOLD (P < 0.01), and Rox (P < 0.05), and the relative decline was not affected by differences in fat mass. CR-induced declines were significant even when Ra and NOLD were normalized to BW or FFM. We conclude that fat mass, like FFM, is a key determinant of protein turnover. However, during CR, higher fat mass does not favorably alter the response of protein metabolism and does not mitigate the loss of FFM. 
Obesity (Silver Spring). 2010 Feb 4. Effects of Adiposity and 30 Days of Caloric Restriction Upon Protein Metabolism in Moderately vs. Severely Obese Women. 

It means that having a high bodyfat % doesn't mean that you wont lose muscle if you diet and that its important to do what you can to keep those muscle, so  do keep your protein high and heavy weights in the gym! If your are very lean you lose a higher percentage of muscle with each kg lost so it gets more and more important the leaner you get.

Tuesday, November 24

A study verifying (again) why you should keep your protein intake during a diet high to keep those hard earned muscle.

PURPOSE:: To examine the influence of dietary protein on lean body mass loss and performance during short-term hypoenergetic weight loss in athletes.

METHODS:: In a parallel design, 20 healthy, young resistance trained athletes were examined for energy expenditure for one week and fed a mixed diet (15% protein, 100% energy) in the second week, followed by a hypoenergetic diet (60% of the habitual energy intake), containing either 15% (~1.0g.kg) protein (control group, n=10; CP) or 35% (~2.3 g.kg) protein (high protein group, n=10; HP) for two weeks. Subjects continued their habitual training throughout the study. Total, lean body and fat mass, performance (squat jump, maximal isometric leg extension, one repetition maximum bench press, muscle endurance bench press and 30 sec wingate test) and fasting blood samples (glucose, non esterified fatty acids (NEFA), glycerol, urea, cortisol, free testosterone, free IGF-1 and growth hormone) and psychological measures were examined at the end of each of the four weeks.

RESULTS:: Total (-3.0 +/- 0.4 kg and -1.5 +/- 0.3 kg for the CP and HP, respectively, p=0.036) and lean body mass loss (-1.6 +/- 0.3 kg and -0.3 +/- 0.3 kg, p=0.006) were significantly larger in the CP compared to the HP. Fat loss, performance and most blood parameters were not influenced by the diet. Urea was higher in HP and NEFA and urea showed a group*time interaction. Fatigue ratings and 'worse than normal' scores on the DALDA were higher in HP.

CONCLUSION:: These results indicate that ~2.3 g.kg-1 or ~35% protein was significantly superior to ~1.0 g.kg-1 or ~15% energy protein for maintenance of lean body mass in healthy young athletes during short-term, hypoenergetic weight loss.


Med Sci Sports Exerc. 2009 Nov 13. Increased Protein Intake Reduces Lean Body Mass Loss during Weight Loss in Athletes.

Tuesday, November 17

I had a great sleep, over 10 hours, still feel tired but not dead tired! Heading to gym today for sure and then I have to move the lawn as we are getting rain and a bit of colder weather! Sooo good!!!!!!!! I’m not a big fan of 40+ days… especially not when the humidity is hight (thunder warnings for Thursday and Friday)
Diet is going well I do like having the last meal around 6-7 as it sort of takes the focus of snacking /eating in front of the TV way, when you have your last meal around 9 its easy to snack on it in front of the TV and that associates eating with TV and then whenever you watch TV you get cravings.

Associative eating is such an easy thing to do, I remember reading somewhere that when this woman was a kid she always got a chocolate bar every time she went to the gas station with her parents, so in adult life she got cravings every time they went to the gas station… hard to resist then wouldn’t it be…

I got word yesterday that the papers for my apartment that I signed a couple of weeks ago have finally arrived! I’m stoked! Its on the way!!!!!! I’m so happy about that! I am so longing for going home that’s its not funny, even had a dream about it last night haha about telling work and everyone’s reactions.

From kollo –
In truth we do not know the relative risk/benefits of giving large amounts of inorganic nitrates to humans. We know that vegetarians (who eat larger amounts of nitrate) have reduced incidence of cancer and vascular disease, and on average live longer than those who eat meat. We believe that nitrate may be a major reason for the beneficial effects of a high vegetable intake, such as reducing blood pressure, protecting the stomach from ulceration, preventing vascular damage, and preventing infection (4). Bryan and colleagues (3) have recently pointed out that the influential "Dietary Approaches to Stop Hypertension" (DASH) diet recommended to hypertensive patients exceeds the WHO ADI for inorganic nitrate by 550%. At the same time, as Derave and Taes (2) point out, it is possible that certain combinations of inorganic nitrate and nitrosatable compounds could be harmful, and that certain individuals, such as those with Barrett's esophagus (8), may be particularly at risk.


We should be aware that there is some slight evidence of risk, but there is also increasing evidence that more nitrate in our diet may be good for us.

http://jap.physiology.org/cgi/conten...7/5/1667?rss=1


Nitrate high food is spinach and beetroot :D So keep those up in your diet and you might be doing yourself some good!

Monday, November 9

Hunger

9-1 Ways to Deal with Hunger on a Diet (ive removed point 7 as i dont want to promote illegal stuff)
the text comeps from Lyle McDonalds site http://www.bodyrecomposition.com/fat-loss/9-ways-to-deal-with-hunger-on-a-diet.html and is a great reed!!!

Diets fail for a lot of reasons but one of the primary ones is simply hunger. I discussed this sort of tangentially in the research review Why Do Obese People not Lose More Weight When Treated with Low-Calorie Diets and one of the comments on that article is what prompted me to write this article.

What is Hunger?
To say that human hunger is complicated is a vast understatement. To cover it in detail would require a series of articles or perhaps an entire book. Research continues to uncover numerous interacting and overlapping hormones (such as leptin, ghrelin, peptide YY, GLP-1 and others) that monitor how much and what someone is eating (along with their body weight) and those all send a signal to the brain that drives a number of processes, not the least of which is hunger.
Now, it would be truly simple if that’s all there was to it but humans also eat/get hungry for non-physiological reasons. We get hungry out of boredom, because we are at a party and it’s expected that we eat, because we just saw a commercial for some food we like and many others.
Simplistically, we might differentiate these different drivers of hunger into physiological and psychological factors although, as I discussed in the article Dieting Psychology vs. Dieting Physiology, the distinction between the two is not only false but increasingly fuzzy. Physiological drives can manifest themselves as ‘psychological’ hunger and psychological factors can affect physiology.
However, even though the distinction is a false one, it is often useful practically to make that division and I”ll be doing so through the rest of that article.

Sufficed to say that human hunger is exceedingly complicated and finding out ways to deal with hunger while dieting is a huge first step in making diets more effective. And with that said, in no particular order of importance, here are 9 Ways to Deal with Hunger on a Diet.

1. Eat More Lean Protein
While dietitians continue to squabble over whether carbohydrates or fats are more filling in the short-term, the data is actually abundantly clear: protein beats them both out. Increasing amounts of research has shown that both acutely and in the long-term, higher protein intakes help blunt hunger. It also helps that, as long as you’re dealing with sources of lean protein (low-fat fish, skinless chicken, even low-fat red meat), it can be tough to get a lot of calories from protein in the first place.
I’d also note that there are many other reasons to consume sufficient amounts of lean protein on a weight loss diet including blood glucose stability and sparing of muscle mass loss. It’s also worth mentioning that a lot of the benefits that are often attributed to ‘low-carbohydrate’ diets have more to do with the increased protein intake; the benefits occur because they are ‘high-protein’.

2. Eat Fruit
For odd reasons fruit has gotten a bad rap for dieting, at least in the athletic and bodybuilding subculture but little could be further from the truth. One aspect of hunger has to do with the status of liver glycogen, when liver glycogen is emptied, a signal is sent to the brain that can stimulate hunger; the corollary is that replenishing liver glycogen tends to make people feel fuller.
The fructose component of fruit works to refill liver glycogen and folks who include a moderate amount of fruit in their weight loss diets often report feeling much less hungry. That’s in addition to the other benefits of fruit (fiber, nutrients). Oh yeah, eat whole fruit, stay away from fruit juice.


3. Eat More Fiber
No list of this sort would be complete without the mention of fiber. Fiber can help with hunger in at least two ways. The first is that the physical ’stretching’ of the stomach is one of many signals about how much food has been eaten; when the stomach is physically stretched the brain thinks you’re full. High-fiber/high-volume foods (e.g. foods that have a lot of volume for few calories) accomplish that most effectively.
Additionally, fiber slows gastric emptying, the rate at which food leave the stomach. By keeping foods in the stomach longer, a high-fiber intake keeps folks full longer. Basically, mom was right, eat your vegetables.

4. Eat (At-Least) Moderate Amounts of Dietary Fat
Ignoring the debate I mentioned above about carbs versus fat and hunger, the simple fact is that exceedingly low-fat diets tend to leave a lot of people hungry in both the short- and long-term. Tying in with my comments about fiber in Number 3, dietary fat also slows gastric emptying (hence the aphorism that high-fat meals really stick to the ribs). While dietary fat does little to blunt hunger in the short-term, moderate intakes tend to keep people fuller longer between meals since the meal sits in the stomach longer.
As well, exceedingly low-fat diets often taste like cardboard, tying into some of the comments I made initially about psychological effects of dieting; people won’t follow a diet that doesn’t taste good for very long. Dietary fat gives food a certain mouth-feel and very low-fat diets remove that, leaving people dissatisfied. The diet usually ends shortly after that.
Research has shown that moderate fat diets improve adherence to dieting and, with rare exceptions, I don’t suggest taking dietary fat much lower than 20-25% of total calories on a fat loss diet. In some cases (such as very low-carbohydrate diets), it may be higher than this.

5. Exercise
I’m hesitant to mention exercise in this article simply because the response to it can vary drastically in terms of hunger control on a diet. Doing the topic justice would take a complete article in and of itself but here I’m going to give a quick overview.
Basically, through myriad overlapping mechanisms, exercise has the potential to increase hunger, decrease hunger or have no effect. Some of the effects are purely physiological. On the one hand, exercise increases leptin transport into the brain which should help some of the other hunger signals work better. On the other hand, some people can get a blood glucose crash with exercise (this is especially true in the early stages of a program) and this can stimulate hunger. Most research suggests that exercise has, if anything, a net benefit in terms of hunger control but it’s even more complicated than that.
Whether or not exercise helps with hunger control ends up interacting with psychological factors that I’m not going to detail here. Some research suggests that people ‘couple’ exercise with their diet. The underlying psychlogy seems to be along the lines of “I exercised today, why would I ruin that by blowing my diet.” That’s good.
However, another category of people often use exercise as an excuse to eat more. The underlying psychology seems to be “I must have burned at least 1000 calories in exercise, I earned that cheeseburger and milkshake.” Of course, since people basically always over-estimate how many calories they burned with exercise, they end up doing more harm than good.
The short-version of this point is this: for some people, regular exercise (and it may not be anything more than a brisk walk) has a profound benefit on keeping them on their diet. And for others it tends to backfire.

6. Consider Intermittent Fasting (IF’ing)
IF’ing is a current dietary trend that, while exact definitions vary, basically refers to a pattern where someone fasts for some portion of the day (perhaps 16-20 hours) and eats most of their food during a short ‘eating period’. Various interpretations are out there but there is emerging research showing a variety of health benefits from this style of eating.
In the context of this article, IF’ing can be particularly valuable for smaller dieters who simply don’t get to eat a lot of food each day. A small female trying to subsist on 1000-1200 calories per day and trying to eat 3-4 times per day is only getting a few small, relatively unsatisfying meals per day.
However, if that same dieter fasts most of the day (many find that hunger goes away after an initial spike in the morning), she can eat 1-2 significantly larger (and more satisfying) meals later in the day.
If you’re interested in IF’ing, I’d direct you to Martin Berkhan’s Leangains.com for the absolute best source of IF information on the net. Martin is currently working on a book on IF’ing and I, for one, can’t wait to see it.

8. Be more Flexible Towards Your Dieting
This is another topic that really deserves a book to fully discuss. I’d say that I need to write that book but the fact is that I already did, the topics I’m going to briefly look at here are discussed in detail in A Guide to Flexible Dieting.
Let me address this topic with a question “What would you do if I told you you could never have something again?” Assume it’s something you like or want, how would you react? Odds are you’d want it that much more, right. It’s human nature, we want what we’re told we can’t have.
Guess what, that’s dieting. Or at least how many dieters approach dieting. Many diets are predicated on some food being bad, off-limits or what have you; dieters go into the diet thinking “I can’t ever eat XXX again in my life” which just makes them want XXX that much more. This is one of the psychological aspects of hunger I mentioned in the introduction.
And, of course, the followup to this is that when dieters do eventually eat XXX (and they will), then they just feel guilty and miserable, figure the diet is blown and eat the entire bag or box of XXX and abandon the diet altogether.
It’s truly a damaging approach to dieting and research has clearly shown that the type of rigid dieter I’m describing above (who expects absolute perfection from their diet or it’s a failure) do worse than more flexible dieters.
The reality is that, within the context of a long-term diet, even small deviations don’t really do much harm (unless the person goes berserk and makes it harmful). That is, say you’re on a diet and you eat a couple hundred calories of cookies because you really wanted them. If you’ve dieted the past 6 days, that’s no big deal. However, if you decide that you are a worthless piece of crap with no willpower and eat another 1000 calories of cookies; well you made it into a problem. Understand?
I always recommend that dieters use strategies like free meals (non-diet meals, preferably eaten out of the house), refeeds (extended periods of deliberate high-carbohydrate over-consumption) and full diet breaks (periods of 10-14 days where the diet is abandoned for maintenance) when they diet. It keeps people from falling into the rigid dieting trap that, invariably, backfires. Again, all of the details can be found in A Guide to Flexible Dieting.

9. Suck it Up or Stay Fat
I want to make it clear that I’m not being facetious with the title of this one; and I’m only being slightly obnoxious. Even if you do everything I talked about above, apply every strategy perfectly, the reality is that you will probably still have some hunger on a diet.
Well…too bad. The simple fact is that losing weight requires eating less than you’re burning and this will, at some point, generate hunger. Now, there are exceptions, extremely overweight individuals often find that they have no appetite in the initial stages of dieting but the reality is that eventually hunger will rear it’s ugly head.
At which point every dieter is faced with a fundamental choice which, put simply is this “What’s more important to me, losing weight, or eating this food?” I’d note that this is also a reason I’m so adamant about the flexible dieting strategies, at least one way of dealing with food cravings is to include them in the diet in a controlled fashion. That way the dieter is controlling the diet, instead of the other way around.
But even with that, hunger is a reality of dieting no matter what else you do. Now, you can try to reframe it (Tom Venuto in his new book suggested telling yourself that “Hunger is fatness leaving the body.”) or you can simply accept it (yes, I know, very Zen) and move on.
But none of that makes the hunger away, it’s just you trying to trick yourself out of feeling bad about it. When that point is reached, there are only two options that I’m going to put very bluntly.
You can suck it up or stay fat.
After you’ve gotten your protein and fruit and fiber and fat and appetite supressants and exercise and flexible dieting strategies down pat, when hunger rears its ugly head, those are the only two options left.

Tuesday, November 3

Now this is interesting

BACKGROUND: Studies of diet and depression have focused primarily on individual nutrients.

AIMS: To examine the association between dietary patterns and depression using an overall diet approach.

METHOD: Analyses were carried on data from 3486 participants (26.2% women, mean age 55.6 years) from the Whitehall II prospective cohort, in which two dietary patterns were identified: 'whole food' (heavily loaded by vegetables, fruits and fish) and 'processed food' (heavily loaded by sweetened desserts, fried food, processed meat, refined grains and high-fat dairy products). Self-reported depression was assessed 5 years later using the Center for Epidemiologic Studies - Depression (CES-D) scale.

RESULTS: After adjusting for potential confounders, participants in the highest tertile of the whole food pattern had lower odds of CES-D depression (OR = 0.74, 95% CI 0.56-0.99) than those in the lowest tertile. In contrast, high consumption of processed food was associated with an increased odds of CES-D depression (OR = 1.58, 95% CI 1.11-2.23).

CONCLUSIONS: In middle-aged participants, a processed food dietary pattern is a risk factor for CES-D depression 5 years later, whereas a whole food pattern is protective.

http://bjp.rcpsych.org/cgi/reprint/195/5/408

Monday, July 27

2 more days of working! Then HOLIDAY (yes –looking forward to it!) wont be updating the blog much. Ill try when we get to Sweden! Im looking at getting a laptop when I’m over there to. A dell I think! Will be good to get a proper keyboard!
If you have any tips let me know, I’m thinking Dell Inspiron 1545 NB Intel Core 2 Duo P8400 2.26GHz / 4GB / 320GB / TFT15.6 / DVDRW / Win Vista Home Premium or a Samsung R510 AS01 Intel Core 2 Duo T5800 2.0GHz / 4096MB / 320GB / TFT15.4 / DVDRW / Win Vista Home Premium
Not sure…… the dell one is a good brand and excellent price! Around 6000 SKR – 1090 AUD.

Went to colonnades yesterday and they had orchid exhibition. Very nice and pretty flowers!





Milk is good for you!
Good to read – especially for women! I take my calcium supplements and eat dariy (cottage cheers/quark and milk) feels good to know its high potency stuff

IJSNEM, 19(1), February 2009. Changes in Body Composition With Yogurt Consumption During Resistance Training in Women.
Resistance training is an effective method to decrease body fat (BF) and increase fat-free mass (FFM) and fat oxidation (FO). Dairy foods containing calcium and vitamin D might enhance these benefits. This study investigated the combined effects of habitual yogurt consumption and resistance training on body composition and metabolism.

Untrained women (N = 35) participated in an 8-wk resistance-training program. The yogurt group (Y) consumed 3 servings of yogurt containing vitamin D per day, and the control groups maintained their baseline low-dairy-calcium diet. Postexercise, Y consumed 1 of the 3 servings/d fat-free yogurt, the protein group consumed an isocaloric product without calcium or vitamin D, and the carbohydrate group consumed an isocaloric product without protein. Strength, body composition, fasted resting metabolic rate (RMR) and FO, and serum 25-hydroxyvitamin D were measured before and after training.

Calories (kcal • kg–1 • d–1) and protein (g • kg–1 • d–1) significantly increased from baseline for Y. FFM increased (main effect p = .002) and %BF decreased (main effect .02) for all groups with training, but Group × Time interactions were not observed. RMR and FO did not change with training for any group.

Habitual consumption of yogurt during resistance training did not augment changes in body composition compared with a low-dairy diet. Y decreased %BF as a result of training, however, even with increased calorie consumption.
http://www.humankinetics.com/ijsnem/...67k6Rk8743b2tv

High-calcium diet can produce weight loss.
Background: Evidence suggests that dietary calcium intake may be inversely related to body weight. One explanatory mechanism is that dietary calcium increases fecal fat excretion, due to either calcium soap formation and/or binding of bile acids (BAs) in the intestine.

Objective: To examine the effect of calcium from low-fat dairy products on fecal fat excretion.
Design: A randomized crossover study with 11 subjects, comparing two 7-d diets: one high in calcium from low-fat dairy products (high-Ca; 2300 mg Ca per d) and one low in calcium (low-Ca; 700 mg Ca per d).
Measurements: All feces were collected during the last 5 days of each diet period and analyzed for fat, energy and calcium content and fatty acid (FA) and BA composition.
Results: Dairy calcium significantly increased the total fecal fat excretion from 5.40.5 g d-1 on the low-Ca diet to 11.51.4 g d-1 on the high-Ca diet (P<0.001). style="font-weight: bold;">Effect of dairy calcium on fecal fat excretion: a randomized crossover trial.
International Journal of Obesity (2008) 32, 1816–1824.
Some studies have shown an inverse relation between calcium intake and body weight or fat mass.

We aimed to investigate the relations of dairy consumption and calcium intake with 6-y changes in body weight and waist circumference (WC).
Multivariate analysis of variance according to dairy consumption or calcium intake quartile was presented, stratified by sex and body weight status at baseline, in 2267 middle-aged French adults.
The associations between dairy products and anthropometric changes differed according to sex and overweight status at baseline. In overweight men only, 6-y changes in weight and WC were inversely associated with the consumption of dairy products—especially that of milk (P = 0.02 for both weight and WC changes) and yogurt (P = 0.01 and 0.03 for weight and WC changes, respectively). No relation was observed with cheese and calcium intake. Positive relations were found between milk consumption and WC change in overweight women and between yogurt consumption and weight change in normal-weight women. Multivariate analyses showed a trend toward increases in weight with high dairy calcium intakes in normal-weight women.
The relation of dairy products and calcium intake with changes in weight and WC may differ according to sex, initial body-weight status, and type of dairy products. The negative association between dairy products and anthropometric changes observed in overweight men was not explained by dairy calcium intakes, which suggests that other components of dairy products or specific dietary patterns associated with dairy consumption may help to explain the observed associations
http://www.ajcn.org/cgi/content/full/88/5/1248

Thursday, July 23

I’m annoyed… yesterday over dinner BF looked at me and said “your eating to much” (this is not the first time he has said it) and I asked him, am I fat? Am I gaming weights. And he goes – no. I’m like –well then I’m not eating to much. He said well compared to me (ehm WTF).. That is one of the most stupid things I have heard. (Lots of other things too – let me just put it this way, next competition he is not helping me that’s for sure)
Rant over

Walking this morning was crisp – ice on the car windows! Atleast its getting lighter outside! Took a great picture!


A very interesting study – especially for women!

High-glycemic load diets have been hypothesized to increase the risk of breast cancer but epidemiologic studies have yielded inconsistent findings. We examined the associations of carbohydrate intake, glycemic index and glycemic load with risk of overall and hormone receptor-defined breast cancer in the Swedish Mammography Cohort, a population-based cohort of 61,433 women who completed a food frequency questionnaire at enrollment in 1987-1990. During a mean follow-up of 17.4 years, we ascertained 2,952 incident cases of invasive breast cancer. Glycemic load but not carbohydrate intake or glycemic index was weakly positively associated with overall breast cancer risk (p for trend = 0.05). In analyses stratified by estrogen receptor (ER) and progesterone receptor (PR) status of the breast tumors, we observed statistically significant positive associations of carbohydrate intake, glycemic index and glycemic load with risk of ER+/PR- breast cancer; the multivariate relative risks comparing extreme quintiles were 1.34 [95% confidence interval (CI) = 0.93-1.94; p for trend = 0.04] for carbohydrate intake, 1.44 (95% CI = 1.06-1.97; p for trend = 0.01) for glycemic index and 1.81 (95% CI = 1.29-2.53; p for trend = 0.0008) for glycemic load. No associations were observed for ER+/PR+ or ER-/PR- breast tumors. These findings suggest that a high carbohydrate intake and diets with high glycemic index and glycemic load may increase the risk of developing ER+/PR- breast cancer.

Susanna C. Larsson, Leif Bergkvist, Alicja Wolk. Glycemic load, glycemic index and breast cancer risk in a prospective cohort of Swedish women. International Journal of Cancer
Volume 125, Issue 1 , Pages153 – 157

(freely translated)
Women that eats a lot of carbohydrates of the wrong kind have a high risk of getting the second most common type of breast cancer
About 20% of all women obtaining breast cancer get the type that is affected by eating habits. A diet high of quick carbohydrates can increase the risk up to 90%
Even a person that eats low carbohydrates but of the wrong kind have a 44% increase in risk.
Carbohydrates to aviod is white bread, potatoes and white rice (I would assume sugar is to be avoided too) Whole grain is to prefer.

The study is done on 61 000 women that answered a questionnaire in 1980 (3000 obtained breast cancer)

Tuesday, July 7

I keep my protein quite high, around 3-4g /body weight and I eat a fair bit of eggs and red meat. This study by Layne Norton and Gabriel Wilson confirms that keeping the protein higher than the EDA of 0.8g/bw and to keep the leucine levels high (leucine is responsible for the anabolic effect in a meal) protein with high levels of leucine can be found in eggs, meats, diary and poultry. They reckon that you need to eat a full meal with high levels of leucine every 3 hour (3g leucine or 0.05g/bw) however I wont change my meal frequency, eating breakky at 7.15 then lunch at 12 then pw at 4 works great for me.. its still interesting and a good study! The study also suggest that one should have carbs with each meal – that’s great (however it doesn’t state what carbs but I’m assuming anything goes) as a lot of people are afraid of carbs.

(whole PDF)
Conclusion
Athletes are more active and have very different goals with regards to nutrition compared to the average person. Athletes looking to maximize muscle mass and strength may benefit from protein levels well above the RDA. While the RDA focuses on minimum needs to achieve nitrogen balance, athletes should focus on consuming sufficient protein to maximize beneficial metabolic outcomes of greater protein intakes on a meal to meal basis. Current research suggests that the amino acid leucine is responsible for much of the anabolic properties of a meal and maximization of mps in response to a meal is dependent upon consuming sufficient leucine (3g or ~0.05g/kg bodyweight) to saturate the mtor signalling pathway. The amount of protein required at a meal to achieve this outcome will differ based on the leucine
content of the protein source with leucine rich protein sources like dairy, egg, meats and poultry being preferable to leucine poor sources of protein such as wheat. These
leucine rich meals should be consumed multiple times per day and consumption of carbohydrate with free form essential amino acids ingested between whole protein
meals may further optimize mps, possibly by overcoming refractoriness.

I have decided to split my training a bit more as I hate doing the legs session and I cheat :-p so new schedule for this week (to see how it works) is
Monday Chest/Tricep
Tuesday Quads/Calfs
Wednesday rest
Thursday Back
Friday Hammys/Abs
Saturday shoulders/bicep
Sundays off
It will make my sessions about 40-45 min long (just enough)

We had 4 degrees outside this morning, I could see my breath inside… the walks becomes very brisk when its like this tho hehe Poor puppies tho, feets on icy cold grass don’t seem very nice…

Friday, July 3

Ah Friday again – now its only 3 weeks and 4 days till I start my holiday and 4 weeks and 5 days till take off! So looking forward to it! (As well as the shopping with the BF in KUL)

BF brought me flowers yesterday, so cute and very pretty! I love lilies and the whole arrangement is so pretty! Looking forward to dinner tonight, potato bake, meat and salad. Going to sleep over at Marias :D Hopefully ill manage to get some shots that are decent ;)

Trained legs, hammy dominant yesterday. Went good, still stuck at the 100kg deads but I think its mainly the upper body bones that’s stopping me. Getting pressed together and hurting…
Found this interesting report on kolo, the power of the mind is a massive thing and the only thing that’s holding you back!

I also bought some new chewies, they look great and actually tastes good too. The flavour stays for a long time :D


I nt J Sports Physiol Perform. 2009 Jun;4(2):244-53. Placebo effects of caffeine on short-term resistance exercise to failure.
Purpose: This study examined the placebo effect of caffeine on number of repetitions (reps), rating of perceived exertion (RPE), blood pressure (BP), and peak heart rate (PHR) during resistance-training exercise with repetitions (reps) performed to volitional failure.

Methods: Following determination of 1-rep maximum in single-leg leg extension, 15 males performed reps to failure at 60% 1-RM in 3 conditions: control, perceived caffeine condition, and perceived placebo condition presented in a randomized order. Participants were informed they would ingest 250 mL of solution that contained either 3 mg.kg-1 caffeine or 3 mg.kg-1placebo 1 h before each exercise trial. A deceptive protocol was employed and subjects consumed a placebo solution in both conditions. During each condition, total reps, RPE for the active muscle and overall body, and PHR were recorded.

Results: Subjects completed 2 more reps when they perceived they had ingested caffeine. RPE was significantly (P = .04) lower in the perceived caffeine and control conditions and RPE for the active muscle was significantly higher across all conditions compared with RPE for the overall body. No substantial differences were evident in PHR across conditions.


Conclusions: Results of this study are similar to studies of actual caffeine ingestion. However, the perception of consuming a substance that purportedly enhances performance is sufficient enough to enable individuals to complete a greater number of reps to failure during short-term resistance exercise.

Wednesday, June 17

Awesome back session yesterday (Im strong and I can see a bit more definition than before) Managed to do 3 sets of dumbbell rows of 32kg (well 70lbs sounds a lot better) seated rows of 150lbs (68kg) I’m pretty much keeping, if not increasing the weights now and that feels great

Hunger – I have a lot of hunger issues, always hungry lol dieting/bulking or maintaining. Its quite annoying. However I do feel a lot better on a slightly higher fat diet but not always. I came across this (at kollo) and its quite interesting – I think it also has to do with the time it takes to eat something. If you take smaller bites then it will take longer to eat (I’m a smart cookie aren’t I lol) and that gives the body more time to register. I know I’m guilty of not taking the time to eat and smaller bites might be a way of achieving that. I also heard somewhere that feeling the texture, not watching tv is a way of dealing with rushing the food down.

This is the study

Background: Food texture plays an important role in food intake regulation. In previous studies we showed a clear effect of viscosity on ad libitum food intake and found indications that eating rate, bite size, and oral processing time (OPT) could play a role.

Objective: The objective was to determine the effect of bite size and OPT of a food on satiation, defined as ad libitum food intake.


Design: Twenty-two healthy subjects participated in all 7 test conditions. Bite sizes were free or fixed to small bite sizes ({approx}5 g) or large bite sizes ({approx}15 g). OPT was free (only in combination with free bite size) or fixed to 3 or 9 s. Subjects consumed chocolate custard through a tube, which was connected to a peristaltic pump. Sound signals indicated OPT duration.


Results: Subjects consumed significantly more when bite sizes were large than when they were small (bite size effect: P < p =" 0.008).">

Conclusion: This study shows that greater oral sensory exposure to a product, by eating with small bite sizes rather than with large bite sizes and increasing OPT, significantly decreases food intake.

Am J Clin Nutr (June 10, 2009). Effect of bite size and oral processing time of a semisolid food on satiation.

Sorry for all the studies I post ;) I do find this interesting!

Coming out from the gym yesterday I saw the sky and it looked awesome, everything was clear except the horizon – very pretty (it was raining where the clouds are – you cant really see it in the photo)


Tuesday, June 16

EEA

Great session yesterday! Benched 3 sets of 50x5! Im very happy!
In regards to training soreness (DOMS), I get sore from every workout, its crazy, in every part, especially my chest and legs. Its quite annoying :p there are some things that one can do to make it better, to get a sports massage, have EEA before and after training – I do this but mainly to boost the protein synthesis and maximise the muscle gains! Warm wraps is a great way to ease the pain and recover too.
massage, warm wraps and EEA

EEA short – EEA is essential amino acids, they contain phenylalanine, isoleucine, leucine, lysin, methionine, threonine, tryptophane and valine. EEA has been proven to stimulate the protein synthesis a lot more than the standards protein shake (whey) and the effect is a lot higher if you take some just before training. The recommendations are to take about 10g before training – the effect has been tested between 6-20 grams but the effect is not increased if you take over 10 grams. (as its quite expensive and tastes like yuk 10 grams is good) its also recommended to take 10 grams after training and then go home and have a proper meal. EEA should also be taken on empty stomach.
EEA is not the building blocks of the muscle they act like the on button to start building, it does count towards your total intake of protein tho. Ie 10g of EEA = 10g of protein.

EEA Timing of intake in relation to exercise
In the experiments described above the amino acids ± glucose were given after exercise. No difference in response was observed when EAA + glucose were given at 1 versus 3 h after exercise (24 ). Similarly, when the same mixture was given immediately after exercise the response of MPS was the same as when given 1 h after exercise (25 ). However, when the EAA/glucose mixture was given immediately before exercise the response was greatly amplified. Not only was the increase during exercise approximately fourfold above the basal value, a (Fig. 5 ). When total area under the curve was calculated, the response was approximately threefold greater when the drink was given after exercise than at rest, and approximately double the response as when ingested after exercise (25 ).

More EEA

so if you want to make the most of your workout (why wouldn't you) hit the EEA - its definitely worth it! I don't take protein shakes anymore - except when i bake with it... only EEA (with a grimace but ah well the taste one have to live with)
Also - only mix the EEA when you drink it as it will start deteriorating when you put water in it and its best to drink it straight away!0

Other than gym and walking the dogs yesterday I didn’t do much. We bought 6 garden lights (well one broke so there is only 5 now) at Bunnings in the weekend and they have been charging for the first couple of days (solar cells) and now they are in the backyard, nice and pretty :)

Hope you have a great day!